Gene Therapy   
Rationale and design of Enhanced Angiogenic Cell Therapy in Acute Myocardial Infarction (ENACT-AMI): the first randomized placebo-controlled trial of enhanced progenitor cell therapy for acute myocardial infarction
Am Heart J, 2010. 159(3): p. 354-60.
M. Taljaard, et al.,
Gene transfer of inducible nitric oxide synthase affords cardioprotection by upregulating heme oxygenase-1 via a nuclear factor-{kappa}B-dependent pathway
Circulation, 2009. 120(13): p. 1222-30.
Q. Li, et al.,
Inhibition of prolyl hydroxylase domain proteins promotes therapeutic revascularization
Circulation, 2009. 120(1): p. 50-9.
C. Loinard, et al.,
Prevention of cardiac dysfunction in acute coxsackievirus B3 cardiomyopathy by inducible expression of a soluble coxsackievirus-adenovirus receptor
Circulation, 2009. 120(23): p. 2358-66.
S. Pinkert, et al.,
Myocardial adeno-associated virus serotype 6-betaARKct gene therapy improves cardiac function and normalizes the neurohormonal axis in chronic heart failure
Circulation, 2009. 119(1): p. 89-98.
G. Rengo, et al.,
Long-term cardiac-targeted RNA interference for the treatment of heart failure restores cardiac function and reduces pathological hypertrophy
Circulation, 2009. 119(9): p. 1241-52.
L. Suckau, et al.,
Hepatocyte growth factor or vascular endothelial growth factor gene transfer maximizes mesenchymal stem cell-based myocardial salvage after acute myocardial infarction
Circulation, 2009. 120(11 Suppl): p. S247-54.
T. Deuse, et al.,
Increased expression of integrin-linked kinase attenuates left ventricular remodeling and improves cardiac function after myocardial infarction
Circulation, 2009. 120(9): p. 764-73.
L. Ding, et al.,
Enhancement of myocardial regeneration through genetic engineering of cardiac progenitor cells expressing Pim-1 kinase
Circulation, 2009. 120(21): p. 2077-87.
K. M. Fischer, et al.,
Novel minicircle vector for gene therapy in murine myocardial infarction
Circulation, 2009. 120(11 Suppl): p. S230-7.
M. Huang, et al.,
Vascular endothelial growth factor-B induces myocardium-specific angiogenesis and arteriogenesis via vascular endothelial growth factor receptor-1- and neuropilin receptor-1-dependent mechanisms
Circulation, 2009. 119(6): p. 845-56.
J. E. Lahteenvuo, et al.,
Epicardial border zone overexpression of skeletal muscle sodium channel SkM1 normalizes activation, preserves conduction, and suppresses ventricular arrhythmia: an in silico, in vivo, in vitro study
Circulation, 2009. 119(1): p. 19-27.
D. H. Lau, et al.,